BNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway-Reference-Cited by-同舟云学术

BNIP3L promotes cardiac fibrosis in cardiac fibroblasts through [Ca2+]i-TGF-β-Smad2/3 pathway

Published:2017-05-15 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Liu Weili,Wang Xinxing,Mei Zhusong,Gong Jingbo,Huang lishuang,Gao Xiujie,Zhao Yun,Ma Jing,Qian Lingjia

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-01936-5.pdf

Reference42 articles.

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2. Nishida, M. Roles of heterotrimeric GTP-binding proteins in the progression of heart failure. Journal of pharmacological sciences 117, 1–5, doi: 10.1254/jphs.11R05CP (2011).

3. Kehat, I. Novel strategies for the treatment of heart failure. Rambam Maimonides medical journal 3, e0011, doi: 10.5041/RMMJ.10078 (2012).

4. Shirani, J., Pick, R., Roberts, W. C. & Maron, B. J. Morphology and significance of the left ventricular collagen network in young patients with hypertrophic cardiomyopathy and sudden cardiac death. Journal of the American College of Cardiology 35, 36–44, doi: 10.1016/S0735-1097(99)00492-1 (2000).

5. Kremneva, L. V. & Abaturova, O. V. Molecular and cellular mechanisms of myocardial remodeling in heart failure. Klinicheskaia meditsina 81, 4–7 (2003).

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