Glatiramer Acetate modulates ion channels expression and calcium homeostasis in B cell of patients with relapsing-remitting multiple sclerosis
Author:
Funder
FISM Fondazione Italiana Sclerosi Multipla 2015/R6
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-38152-8.pdf
Reference39 articles.
1. Weiner, H. L. Multiple Sclerosis Is an Inflammatory T-Cell–Mediated Autoimmune Disease. Arch. Neurol. 61, 1613–1615 (2004).
2. Pröbstel, A. K., Sanderson, N. S. & Derfuss, T. B Cells and Autoantibodies in Multiple Sclerosis. Int. J. Mol. Sci. 16, 16576–16592 (2015).
3. Claes, N., Fraussen, J., Stinissen, P., Hupperts, R. & Somers, V. B Cells Are Multifunctional Players in Multiple Sclerosis Pathogenesis: Insights from Therapeutic Interventions. Front. Immunol. 6, 642 (2015).
4. VJohnson, K. P. Glatiramer acetate for treatment of relapsing-remitting multiple sclerosis. Expert. Rev. Neurother. 12, 371–384 (2012).
5. Ireland, S. J. et al. The effect of glatiramer acetate therapy on functional properties of B cells from patients with relapsing-remitting multiple sclerosis. JAMA Neurol 71, 1421–1428 (2014).
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