Two-fold elevation of endogenous GDNF levels in mice improves motor coordination without causing side-effects-Reference-Cited by-同舟云学术

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Two-fold elevation of endogenous GDNF levels in mice improves motor coordination without causing side-effects

Published:2018-08-08 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Mätlik Kärt,Võikar Vootele^ORCID,Vilenius Carolina,Kulesskaya Natalia,Andressoo Jaan-Olle^ORCID

Funder

Suomen Akatemia (Academy of Finland)

EC | Horizon 2020 (European Union Framework Programme for Research and Innovation)

University of Helsinki, Faculty of Medicine and Helsinki Institute of Life Science

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-29988-1.pdf

Reference74 articles.

1. Lin, L. F., Doherty, D. H., Lile, J. D., Bektesh, S. & Collins, F. GDNF: a glial cell line-derived neurotrophic factor for midbrain dopaminergic neurons. Science 260, 1130–1132, https://doi.org/10.1126/science.8493557 (1993).

2. Hoffer, B. J. et al. Glial cell line-derived neurotrophic factor reverses toxin-induced injury to midbrain dopaminergic neurons in vivo. Neurosci Lett 182, 107–111, https://doi.org/10.1016/0304-3940(94)90218-6 (1994).

3. Georgievska, B., Kirik, D. & Bjorklund, A. Aberrant sprouting and downregulation of tyrosine hydroxylase in lesioned nigrostriatal dopamine neurons induced by long-lasting overexpression of glial cell line derived neurotrophic factor in the striatum by lentiviral gene transfer. Exp Neurol 177, 461–474, https://doi.org/10.1006/exnr.2002.8006 (2002).

4. Georgievska, B., Kirik, D. & Bjorklund, A. Overexpression of glial cell line-derived neurotrophic factor using a lentiviral vector induces time- and dose-dependent downregulation of tyrosine hydroxylase in the intact nigrostriatal dopamine system. J Neurosci 24, 6437–6445, https://doi.org/10.1523/JNEUROSCI.1122-04.2004 (2004).

5. Meissner, W. G. et al. Priorities in Parkinson’s disease research. Nat Rev Drug Discov 10, 377–393, https://doi.org/10.1038/nrd3430 (2011).

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