PLK2 Plays an Essential Role in High D-Glucose-Induced Apoptosis, ROS Generation and Inflammation in Podocytes
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-00686-8.pdf
Reference53 articles.
1. Du, P. et al. NOD2 promotes renal injury by exacerbating inflammation and podocyte insulin resistance in diabetic nephropathy. Kidney Int 84, 265–276 (2013).
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3. Susztak, K., Raff, A. C., Schiffer, M. & Böttinger, E. P. Glucose-induced reactive oxygen species cause apoptosis of podocytes and podocyte depletion at the onset of diabetic nephropathy. Diabetes 55, 225–233 (2006).
4. Lewko, B. & Stepinski, J. Hyperglycemia and mechanical stress: targeting the renal podocyte. J Cell Physiol 221, 288–95 (2009).
5. Welsh, G. I. & Coward, R. J. Podocytes, glucose and insulin. Curr Opin Nephrol Hypertensn 19, 379–84 (2010).
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