Usp16 modulates Wnt signaling in primary tissues through Cdkn2a regulation-Reference-Cited by-同舟云学术

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Usp16 modulates Wnt signaling in primary tissues through Cdkn2a regulation

Published:2018-11-30 Issue:1 Volume:8 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Adorno Maddalena,di Robilant Benedetta Nicolis,Sikandar Shaheen Shabbir,Acosta Veronica Haro,Antony Jane^ORCID,Heller Craig H.,Clarke Michael F.

Funder

CZ Biohub LumindRDS foundation

AIRC and Marie Curie action

U.S. Department of Defense

LuMind RDS foundation

CZ Biohub Luwing foundation

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-018-34562-w.pdf

Reference41 articles.

1. Clevers, H. & Nusse, R. Wnt/beta-catenin signaling and disease. Cell 149, 1192–1205 (2012).

2. Kalani, M. Y. et al. Wnt-mediated self-renewal of neural stem/progenitor cells. Proc Natl Acad Sci USA 105, 16970–16975 (2008).

3. Hofmann, J. W., McBryan, T., Adams, P. D. & Sedivy, J. M. The effects of aging on the expression of Wnt pathway genes in mouse tissues. Age (Dordr) 36, 9618 (2014).

4. Ye, X. et al. Downregulation of Wnt signaling is a trigger for formation of facultative heterochromatin and onset of cell senescence in primary human cells. Mol Cell 27, 183–196 (2007).

5. Manolagas, S. C. & Almeida, M. Gone with the Wnts: beta-catenin, T-cell factor, forkhead box O, and oxidative stress in age-dependent diseases of bone, lipid, and glucose metabolism. Mol. Endocrinol. 21, 2605–2614 (2007).

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