Mechanisms of fitness cost reduction for rifampicin-resistant strains with deletion or duplication mutations in rpoB
Author:
Funder
Vetenskapsrådet
Carl Tryggers Stiftelse för Vetenskaplig Forskning
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-36005-y.pdf
Reference20 articles.
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2. Brandis, G., Pietsch, F., Alemayehu, R. & Hughes, D. Comprehensive phenotypic characterization of rifampicin resistance mutations in Salmonella provides insight into the evolution of resistance in Mycobacterium tuberculosis. J Antimicrob Chemother 70, 680–685 (2015).
3. Ho, M. X., Hudson, B. P., Das, K., Arnold, E. & Ebright, R. H. Structures of RNA polymerase–antibiotic complexes. Curr Opin Struc Biol 19, 715–723 (2009).
4. Brandis, G., Wrande, M., Liljas, L. & Hughes, D. Fitness-compensatory mutations in rifampicin-resistant RNA polymerase. Mol Microbiol 85, 142–151 (2012).
5. Brandis, G. & Hughes, D. Genetic characterization of compensatory evolution in strains carrying rpoB Ser531Leu, the rifampicin resistance mutation most frequently found in clinical isolates. J Antimicrob Chemother 68, 2493–2497 (2013).
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