α-Synuclein Amyloids Hijack Prion Protein to Gain Cell Entry, Facilitate Cell-to-Cell Spreading and Block Prion Replication-Reference-Cited by-同舟云学术

α-Synuclein Amyloids Hijack Prion Protein to Gain Cell Entry, Facilitate Cell-to-Cell Spreading and Block Prion Replication

Published:2017-08-30 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Aulić Suzana,Masperone Lara,Narkiewicz Joanna,Isopi Elisa,Bistaffa Edoardo,Ambrosetti Elena,Pastore Beatrice,De Cecco Elena,Scaini Denis,Zago Paola,Moda Fabio,Tagliavini Fabrizio,Legname Giuseppe^ORCID

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-10236-x.pdf

Reference44 articles.

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2. McCann, H., Stevens, C. H., Cartwright, H. & Halliday, G. M. alpha-Synucleinopathy phenotypes. Parkinsonism Relat Disord 20(Suppl 1), S62–67, doi: 10.1016/s1353-8020(13)70017-8 (2014).

3. Li, J. Y. et al. Lewy bodies in grafted neurons in subjects with Parkinson’s disease suggest host-to-graft disease propagation. Nat Med 14, 501–503, doi: 10.1038/nm1746 (2008).

4. Kordower, J. H., Chu, Y., Hauser, R. A., Freeman, T. B. & Olanow, C. W. Lewy body-like pathology in long-term embryonic nigral transplants in Parkinson’s disease. Nat Med 14, 504–506, doi: 10.1038/nm1747 (2008).

5. Desplats, P. et al. Inclusion formation and neuronal cell death through neuron-to-neuron transmission of alpha-synuclein. Proc Natl Acad Sci USA 106, 13010–13015, doi: 10.1073/pnas.0903691106 (2009).

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