Reversal of stress fibre formation by Nitric Oxide mediated RhoA inhibition leads to reduction in the height of preformed thrombi
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-21167-6.pdf
Reference43 articles.
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2. Radomski, M. W., Palmer, R. M. J. & Moncada, S. Comparative Pharmacology of Endothelium-Derived Relaxing Factor, Nitric-Oxide and Prostacyclin in Platelets. Brit J Pharmacol 92, 181–187 (1987).
3. Hollopeter, G. et al. Identification of the platelet ADP receptor targeted by antithrombotic drugs. Nature 409, 202–207, https://doi.org/10.1038/35051599 (2001).
4. Remijn, J. A. et al. Role of ADP receptor P2Y(12) in platelet adhesion and thrombus formation in flowing blood. Arterioscler Thromb Vasc Biol 22, 686–691 (2002).
5. Boo, Y. C. & Jo, H. Flow-dependent regulation of endothelial nitric oxide synthase: role of protein kinases. Am J Physiol Cell Physiol 285, C499–508, https://doi.org/10.1152/ajpcell.00122.2003 (2003).
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