Dramatic increases in blood glutamate concentrations are closely related to traumatic brain injury-induced acute lung injury
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-05574-9.pdf
Reference44 articles.
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2. Oddo, M. et al. Acute lung injury is an independent risk factor for brain hypoxia after severe traumatic brain injury. Neurosurgery 67, 338–344 (2010).
3. Arbour, R. B. Traumatic brain injury: pathophysiology, monitoring, and mechanism-based care. Crit. Care Nurs. Clin. North Am. 25, 297–319 (2013).
4. Palmer, A. M. et al. Traumatic brain injury-induced excitotoxicity assessed in a controlled cortical impact model. J. Neurochem. 61, 2015–2024 (1993).
5. Chamoun, R., Suki, D., Gopinath, S. P., Goodman, J. C. & Robertson, C. Role of extracellular glutamate measured by cerebral microdialysis in severe traumatic brain injury. J. Neurosurg. 113, 564–570 (2010).
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