Gingerol suppresses sepsis-induced acute kidney injury by modulating methylsulfonylmethane and dimethylamine production
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-30522-6.pdf
Reference54 articles.
1. Coldewey, S. M. et al. Erythropoietin attenuates acute kidney dysfunction in murine experimental sepsis by activation of the β-common receptor. Kidney Int. 84, 482–90 (2013).
2. Umbro, I., Gentile, G., Tinti, F., Muiesan, P. & Mitterhofer, A. P. Recent advances in pathophysiology and biomarkers of sepsis-induced acute kidney injury. J. Infec. 72, 131–142 (2016).
3. Seija, M. et al. Role of peroxynitrite in sepsis-induced acute kidney injury in an experimental model of sepsis in rats. Shock. 38, 403–410 (2012).
4. Rodrigues, C. E. et al. Effects of continuous erythropoietin receptor activator in sepsis-induced acute kidney injury and multi-organ dysfunction. PLoS One. 7, e29893 (2012).
5. Boffa, J. J. & Arendshorst, W. J. Maintenance of renal vascular reactivity contributes to acute renal injury during endotoxemic shock. J. Am. Soc. Nephrol. 16, 117–124 (2005).
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