Sensory primary cilium is a responsive cAMP microdomain in renal epithelia-Reference-Cited by-同舟云学术

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Sensory primary cilium is a responsive cAMP microdomain in renal epithelia

Published:2019-04-25 Issue:1 Volume:9 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Sherpa Rinzhin T.,Mohieldin Ashraf M.,Pala Rajasekharreddy,Wachten Dagmar,Ostrom Rennolds S.,Nauli Surya M.

Funder

United States Department of Defense | United States Army | Army Medical Command | Congressionally Directed Medical Research Programs

U.S. Department of Health & Human Services | NIH | National Heart, Lung, and Blood Institute

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-019-43002-2.pdf

Reference86 articles.

1. Pazour, G. J. et al. Chlamydomonas IFT88 and its mouse homologue, polycystic kidney disease gene tg737, are required for assembly of cilia and flagella. J. Cell Biol. 151, 709–18 (2000).

2. Nauli, S. M. Loss of Polycystin-1 in Human Cyst-Lining Epithelia Leads to Ciliary Dysfunction. J. Am. Soc. Nephrol. 17, 1015–1025 (2006).

3. Vassilev, P. M. et al. Polycystin-2 is a novel cation channel implicated in defective intracellular Ca2+ homeostasis in polycystic kidney disease. Biochem. Biophys. Res. Commun. 282, 341–50 (2001).

4. Xu, C. et al. Human ADPKD primary cyst epithelial cells with a novel, single codon deletion in the PKD1 gene exhibit defective ciliary polycystin localization and loss of flow-induced Ca2+ signaling. AJP Ren. Physiol. 292, F930–F945 (2006).

5. Nauli, S. M. et al. Endothelial Cilia Are Fluid Shear Sensors That Regulate Calcium Signaling and Nitric Oxide Production Through Polycystin-1. Circulation 117, 1161–1171 (2008).

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