PID1 increases chemotherapy-induced apoptosis in medulloblastoma and glioblastoma cells in a manner that involves NFκB
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-00947-6.pdf
Reference33 articles.
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2. Zhang, C. M. et al. Over-expression of NYGGF4 inhibits glucose transport in 3T3-L1 adipocytes via attenuated phosphorylation of IRS-1 and Akt. Acta pharmacologica Sinica 30, 120–124, doi: 10.1038/aps.2008.9 (2009).
3. Zhao, Y. et al. Overexpression of NYGGF4 (PID1) induces mitochondrial impairment in 3T3-L1 adipocytes. Mol. Cell. Biochem. 340, 41–48, doi: 10.1007/s11010-010-0398-5 (2010).
4. Wu, W. L. et al. Over-expression of NYGGF4 (PID1) inhibits glucose transport in skeletal myotubes by blocking the IRS1/PI3K/AKT insulin pathway. Mol. Genet. Metab. 102, 374–377, doi: 10.1016/j.ymgme.2010.11.165 (2011).
5. Kajiwara, Y. et al. Extensive proteomic screening identifies the obesity-related NYGGF4 protein as a novel LRP1-interactor, showing reduced expression in early Alzheimer’s disease. Mol Neurodegener 5, 1–11, doi: 10.1186/1750-1326-5-1 (2010).
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Dual role of PID1 in regulating apoptosis induced by distinct anticancer-agents through AKT/Raf-1-dependent pathway in hepatocellular carcinoma;Cell Death Discovery;2023-04-28
2. IGF2BP2 Induces U251 Glioblastoma Cell Chemoresistance by Inhibiting FOXO1-Mediated PID1 Expression Through Stabilizing lncRNA DANCR;Frontiers in Cell and Developmental Biology;2022-01-24
3. GRK2 promotes growth of medulloblastoma cells and protects them from chemotherapy-induced apoptosis;Scientific Reports;2019-09-25
4. Overexpression of PID1 reduces high density lipoprotein level and functionality in swine;IUBMB Life;2019-07-24
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