Epithelial Chloride Transport by CFTR Requires TMEM16A-Reference-Cited by-同舟云学术

Epithelial Chloride Transport by CFTR Requires TMEM16A

Published:2017-09-29 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Benedetto Roberta,Ousingsawat Jiraporn,Wanitchakool Podchanart,Zhang Yong,Holtzman Michael J.^ORCID,Amaral Margarida^ORCID,Rock Jason R.,Schreiber Rainer,Kunzelmann Karl

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/s41598-017-10910-0.pdf

Reference49 articles.

1. Riordan, J. R. CFTR function and prospects for therapy. Annual review of biochemistry 77, 701–726 (2008).

2. Pedemonte, N. & Galietta, L. J. Structure and Function of TMEM16 Proteins (Anoctamins). Physiol Rev 94, 419–459 (2014).

3. Kunzelmann, K. & Mall, M. Electrolyte transport in the colon: Mechanisms and implications for disease. Physiological Reviews 82, 245–289 (2002).

4. Ousingsawat, J. et al. Loss of TMEM16A causes a defect in epithelial Ca2+ dependent chloride transport. J Biol Chem 284, 28698–28703 (2009).

5. Huang, F. et al. Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction. Proc. Natl. Acad. Sci USA 109, 16354–16359 (2012).

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