Epithelial Chloride Transport by CFTR Requires TMEM16A
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-10910-0.pdf
Reference49 articles.
1. Riordan, J. R. CFTR function and prospects for therapy. Annual review of biochemistry 77, 701–726 (2008).
2. Pedemonte, N. & Galietta, L. J. Structure and Function of TMEM16 Proteins (Anoctamins). Physiol Rev 94, 419–459 (2014).
3. Kunzelmann, K. & Mall, M. Electrolyte transport in the colon: Mechanisms and implications for disease. Physiological Reviews 82, 245–289 (2002).
4. Ousingsawat, J. et al. Loss of TMEM16A causes a defect in epithelial Ca2+ dependent chloride transport. J Biol Chem 284, 28698–28703 (2009).
5. Huang, F. et al. Calcium-activated chloride channel TMEM16A modulates mucin secretion and airway smooth muscle contraction. Proc. Natl. Acad. Sci USA 109, 16354–16359 (2012).
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