Promoter Usage and Dynamics in Vascular Smooth Muscle Cells Exposed to Fibroblast Growth Factor-2 or Interleukin-1β
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-30702-4.pdf
Reference50 articles.
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2. Lindner, V. & Reidy, M. A. Proliferation of Smooth-Muscle Cells after Vascular Injury Is Inhibited by an Antibody against Basic Fibroblast Growth-Factor. Proceedings of the National Academy of Sciences of the United States of America 88, 3739–3743, https://doi.org/10.1073/pnas.88.9.3739 (1991).
3. Kinsella, M. G., Irvin, C., Reidy, M. A. & Wight, T. N. Removal of heparan sulfate by heparinase treatment inhibits FGF-2-dependent smooth muscle cell proliferation in injured rat carotid arteries. Atherosclerosis 175, 51–57, https://doi.org/10.1016/j.atherosclerosis.2004.01.045 (2004).
4. Francis, D. J. et al. Blockade of vascular smooth muscle cell proliferation and intimal thickening after balloon injury by the sulfated oligosaccharide PI-88: phosphomannopentaose sulfate directly binds FGF-2, blocks cellular signaling, and inhibits proliferation. Circulation research 92, e70–77, https://doi.org/10.1161/01.RES.0000071345.76095.07 (2003).
5. Raj, T. et al. Inhibition of fibroblast growth factor receptor signaling attenuates atherosclerosis in apolipoprotein E-deficient mice. Arteriosclerosis, thrombosis, and vascular biology 26, 1845–1851, https://doi.org/10.1161/01.ATV.0000227689.41288.5e (2006).
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