Increased LIGHT expression and activation of non-canonical NF-κB are observed in gastric lesions of MyD88-deficient mice upon Helicobacter felis infection
Author:
Funder
U.S. Department of Health & Human Services | NIH | National Cancer Institute
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-019-43417-x.pdf
Reference38 articles.
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2. Ernst, M., Thiem, S., Nguyen, P. M., Eissmann, M. & Putoczki, T. L. Epithelial gp130/Stat3 functions: an intestinal signaling node in health and disease. Semin Immunol 26, 29–37, https://doi.org/10.1016/j.smim.2013.12.006 (2014).
3. Menheniott, T. R., Judd, L. M. & Giraud, A. S. STAT3: a critical component in the response to Helicobacter pylori infection. Cellular microbiology 17, 1570–1582, https://doi.org/10.1111/cmi.12518 (2015).
4. Jenkins, B. J. et al. Hyperactivation of Stat3 in gp130 mutant mice promotes gastric hyperproliferation and desensitizes TGF-beta signaling. Nature medicine 11, 845–852, https://doi.org/10.1038/nm1282 (2005).
5. Backert, S. & Naumann, M. What a disorder: proinflammatory signaling pathways induced by Helicobacter pylori. Trends in microbiology 18, 479–486, https://doi.org/10.1016/j.tim.2010.08.003 (2010).
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