Campylobacter jejuni bile exposure influences outer membrane vesicles protein content and bacterial interaction with epithelial cells
Author:
Funder
Carl Trygger Foundation Foundation for Medical Research (Insamlingsstiftelsen) at Umeå University
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-35409-0.pdf
Reference78 articles.
1. Bernstein, H. et al. Activation of the promoters of genes associated with DNA damage, oxidative stress, ER stress and protein malfolding by the bile salt, deoxycholate. Toxicology letters 108, 37–46 (1999).
2. Sistrunk, J. R., Nickerson, K. P., Chanin, R. B., Rasko, D. A. & Faherty, C. S. Survival of the Fittest: How Bacterial Pathogens Utilize Bile To Enhance Infection. Clinical microbiology reviews 29, 819–836, https://doi.org/10.1128/cmr.00031-16 (2016).
3. Jia, W. & Xie, G. Probiotics, bile acids and gastrointestinal carcinogenesis. Nature reviews. Gastroenterology & hepatology 15, 205, https://doi.org/10.1038/nrgastro.2018.24 (2018).
4. Joyce, S. A. & Gahan, C. G. Bile Acid Modifications at the Microbe-HostInterface: Potential for Nutraceutical and Pharmaceutical Interventions in Host Health. Annual review of food science and technology 7, 313–333, https://doi.org/10.1146/annurev-food-041715-033159 (2016).
5. Kirkpatrick, B. D. & Tribble, D. R. Update on human Campylobacter jejuni infections. Curr Opin Gastroenterol 27, 1–7, https://doi.org/10.1097/MOG.0b013e3283413763 (2011).
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