A cold-inducible RNA-binding protein (CIRP)-derived peptide attenuates inflammation and organ injury in septic mice
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-017-13139-z.pdf
Reference64 articles.
1. Singer, M. et al. The third international consensus definitions for sepsis and septic shock (Sepsis-3). JAMA 315, 801–810, https://doi.org/10.1001/jama.2016.0287 (2016).
2. Shankar-Hari, M. et al. Developing a new definition and assessing new clinical criteria for septic shock: for the Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3). JAMA 315, 775–787, https://doi.org/10.1001/jama.2016.0289 (2016).
3. Seymour, C. W. et al. Assessment of clinical criteria for cepsis: for the Third International Consensus Definitions for Sepsis and Septic Shock (Sepsis-3). JAMA 315, 762–774, https://doi.org/10.1001/jama.2016.0288 (2016).
4. Fleischmann, C. et al. Assessment of global incidence and mortality of hospital-treated sepsis. Current estimates and limitations. Am J Respir Crit Care Med 193, 259–272, https://doi.org/10.1164/rccm.201504-0781OC (2016).
5. Hall, M. J., Williams, S. N., DeFrances, C. J. & Golosinskiy, A. 1–8 (National Center for Health Statistics Hyattsville, MD, 2011).
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1. miR-485-5p acts as a negative regulator of sepsis-associated acute kidney injury through targeting ICAM-1;2024-09-02
2. Extracellular CIRP promotes Kupffer cell inflammatory polarization in sepsis;Frontiers in Immunology;2024-05-30
3. Regulation of Alternative Splicing of Lipid Metabolism Genes in Sepsis-Induced Liver Damage by RNA-Binding Proteins;Inflammation;2024-05-09
4. CIRP increases Foxp3+ regulatory T cells and inhibits development of Th17 cells by enhancing TLR4-IL-2 signaling in the late phase of sepsis;International Immunopharmacology;2024-05
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