Proinsulin-mediated induction of type 1 diabetes in HLA-DR4-transgenic mice
Author:
Funder
The work was funded by UCB Pharma Ltd
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/s41598-018-32546-4.pdf
Reference13 articles.
1. Pow Sang, L., Surls, J., Mendoza, M., Casares, S. & Brumeanu, T. HLA-DR*0401 expression in the NOD mice prevents the development of autoimmune diabetes by multiple alterations in the T-cell compartment. Cell Immunol 298, 54–65, https://doi.org/10.1016/j.cellimm.2015.09.003 (2015).
2. Wen, L., Chen, N. Y., Tang, J., Sherwin, R. & Wong, F. S. The regulatory role of DR4 in a spontaneous diabetes DQ8 transgenic model. J Clin Invest 107, 871–880, https://doi.org/10.1172/JCI11708 (2001).
3. Gebe, J. A. et al. Age-dependent loss of tolerance to an immunodominant epitope of glutamic acid decarboxylase in diabetic-prone RIP-B7/DR4 mice. Clin Immunol 121, 294–304, https://doi.org/10.1016/j.clim.2006.08.002 (2006).
4. Narendran, P., Mannering, S. I. & Harrison, L. C. Proinsulin-a pathogenic autoantigen in type 1 diabetes. Autoimmun Rev 2, 204–210 (2003).
5. Zhang, L., Nakayama, M. & Eisenbarth, G. S. Insulin as an autoantigen in NOD/human diabetes. Curr Opin Immunol 20, 111–118, https://doi.org/10.1016/j.coi.2007.11.005 (2008).
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