Tsc1 deficiency impairs mammary development in mice by suppression of AKT, nuclear ERα and cell-cycle-driving proteins-Reference-Cited by-同舟云学术

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Tsc1 deficiency impairs mammary development in mice by suppression of AKT, nuclear ERα and cell-cycle-driving proteins

Published:2016-01-22 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Qin Zhenqi,Zheng Hang,Zhou Ling,Ou Yanhua,Huang Bin,Yan Bo,Qin Zhenshu,Yang Cuilan,Su Yongchun,Bai Xiaochun,Guo Jiasong,Lin Jun

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep19587.pdf

Reference43 articles.

1. Orlova, K. A. & Crino, P. B. The tuberous sclerosis complex. Ann N Y Acad Sci. 1184, 87–105 (2010).

2. van Slegtenhorst, M. et al. Interaction between hamartin and tuberin, the TSC1 and TSC2 gene products. Hum Mol Genet. 7, 1053–1057 (1998).

3. Wienecke, R., Konig, A. & DeClue, J. E. Identification of tuberin, the tuberous sclerosis-2 product. Tuberin possesses specific Rap1GAP activity. J. Biol Chem. 270, 16409–16414 (1995).

4. Benvenuto, G. et al. The tuberous sclerosis-1 (TSC1) gene product hamartin suppresses cell growth and augments the expression of the TSC2 product tuberin by inhibiting its ubiquitination. Oncogene. 19, 6306–6316 (2000).

5. Chong-Kopera, H. et al. TSC1 stabilizes TSC2 by inhibiting the interaction between TSC2 and the HERC1 ubiquitin ligase. J Biol Chem. 281, 8313–8316 (2006).

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