A self-reinforcing cycle hypothesis in heart failure pathogenesis
Author:
Funder
Gouvernement du Canada | Natural Sciences and Engineering Research Council of Canada
Publisher
Springer Science and Business Media LLC
Link
https://www.nature.com/articles/s44161-024-00480-6.pdf
Reference100 articles.
1. Savarese, G. et al. Global burden of heart failure: a comprehensive and updated review of epidemiology. Cardiovasc. Res. 118, 3272–3287 (2023).
2. Neubauer, S. The failing heart—an engine out of fuel. N. Engl. J. Med. 356, 1140–1151 (2007). Critical appraisal of the cardiac inefficiency model of heart failure with a focus on the interactions between the cardiac mitochondrial OXPHOS pathway and the phosphagen system involving creatine.
3. Lopaschuk, G. D., Karwi, Q. G., Tian, R., Wende, A. R. & Abel, E. D. Cardiac energy metabolism in heart failure. Circ. Res. 128, 1487–1513 (2021).
4. Packer, M., Lam, C. S. P., Lund, L. H., Maurer, M. S. & Borlaug, B. A. Characterization of the inflammatory–metabolic phenotype of heart failure with a preserved ejection fraction: a hypothesis to explain influence of sex on the evolution and potential treatment of the disease. Eur. J. Heart Fail. 22, 1551–1567 (2020).
5. Decherd, G. & Visscher, M. B. Energy metabolism of the failing heart. J. Exp. Med. 59, 195–199 (1934). A cardiac inefficiency model is first proposed to explain heart failure.
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