Functional defects in CD4+ CD25high FoxP3+ regulatory cells in ankylosing spondylitis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Multidisciplinary
Link
http://www.nature.com/articles/srep37559.pdf
Reference41 articles.
1. Brown, M. A., Kenna, T. & Wordsworth, B. P. Genetics of ankylosing spondylitis-insights into pathogenesis. Nature reviews. Rheumatology 12, 81–91 (2016).
2. Cortes, A. et al. Identification of multiple risk variants for ankylosing spondylitis through high-density genotyping of immune-related loci. Nature genetics 45, 730–738 (2013).
3. Chen, L. et al. Silencing or inhibition of endoplasmic reticulum aminopeptidase 1 (ERAP1) suppresses free heavy chain expression and Th17 responses in ankylosing spondylitis. Annals of the rheumatic diseases 75, 916–923 (2016).
4. Walter, G. J. et al. Interaction with activated monocytes enhances cytokine expression and suppressive activity of human CD4+CD45ro+CD25+CD127(low) regulatory T cells. Arthritis and rheumatism 65, 627–638 (2013).
5. Wang, C., Liao, Q., Hu, Y. & Zhong, D. T lymphocyte subset imbalances in patients contribute to ankylosing spondylitis. Experimental and therapeutic medicine 9, 250–256 (2015).
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