No contribution of angiotensin-converting enzyme (ACE) gene variants to severe obesity: a model for comprehensive case/control and quantitative cladistic analysis of ACE in human diseases-Reference-Cited by-同舟云学术

No contribution of angiotensin-converting enzyme (ACE) gene variants to severe obesity: a model for comprehensive case/control and quantitative cladistic analysis of ACE in human diseases

Published:2006-12-13 Issue:3 Volume:15 Page:320-327
ISSN:1018-4813
Container-title:European Journal of Human Genetics
language:en
Short-container-title:Eur J Hum Genet

Author:

Bell Christopher G,Meyre David,Petretto Enrico,Levy-Marchal Claire,Hercberg Serge,Charles Marie Aline,Boyle Cliona,Weill Jacques,Tauber Maïte,Mein Charles A,Aitman Timothy J,Froguel Philippe,Walley Andrew J

Publisher

Springer Science and Business Media LLC

Subject

Genetics (clinical),Genetics

Link

http://www.nature.com/articles/5201754.pdf

Reference47 articles.

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3. Bell CG, Walley AJ, Froguel P : The genetics of human obesity. Nat Rev Genet 2005; 6: 221–234.

4. Meyre D, Bouatia-Naji N, Tounian A et al: Variants of ENPP1 are associated with childhood and adult obesity and increase the risk of glucose intolerance and type 2 diabetes. Nat Genet 2005; 37: 863–867.

5. Vasseur F, Helbecque N, Lobbens S et al: Hypoadiponectinaemia and high risk of type 2 diabetes are associated with adiponectin-encoding (ACDC) gene promoter variants in morbid obesity: evidence for a role of ACDC in diabesity. Diabetologia 2005; 48: 892–899.

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