Expanded high-resolution genetic study of 109 Swedish families with Alzheimer's disease-Reference-Cited by-同舟云学术

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Expanded high-resolution genetic study of 109 Swedish families with Alzheimer's disease

Published:2007-10-24 Issue:2 Volume:16 Page:202-208
ISSN:1018-4813
Container-title:European Journal of Human Genetics
language:en
Short-container-title:Eur J Hum Genet

Author:

Sillén Anna,Andrade Jorge,Lilius Lena,Forsell Charlotte,Axelman Karin,Odeberg Jacob,Winblad Bengt,Graff Caroline

Publisher

Springer Science and Business Media LLC

Subject

Genetics(clinical),Genetics

Link

http://www.nature.com/articles/5201946.pdf

Reference51 articles.

1. Selkoe DJ : Cell biology of the amyloid beta-protein precursor and the mechanism of Alzheimer's disease. Annu Rev Cell Biol 1994; 10: 373–403.

2. Haass C, Selkoe DJ : Cellular processing of beta-amyloid precursor protein and the genesis of amyloid beta-peptide. Cell 1993; 75: 1039–1042.

3. Hardy JA, Higgins GA : Alzheimer's disease: the amyloid cascade hypothesis. Science 1992; 256: 184–185.

4. Goate A, Chartier-Harlin M-C, Mullan M et al: Segregation of a missense mutation in the amyloid precursor protein gene with familial Alzheimer's disease. Nature 1991; 349: 704–706.

5. Rovelet-Lecrux A, Hannequin D, Raux G et al: APP locus duplication causes autosomal dominant early-onset Alzheimer disease with cerebral amyloid angiopathy. Nat Genet 2006; 38: 24–26.

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5. Genomic variants, genes, and pathways of Alzheimer's disease: An overview;American Journal of Medical Genetics Part B: Neuropsychiatric Genetics;2016-12-12

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