A trio family study showing association of the lymphotoxin-α N26 (804A) allele with coronary artery disease
Author:
Publisher
Springer Science and Business Media LLC
Subject
Genetics (clinical),Genetics
Link
http://www.nature.com/articles/5201244.pdf
Reference22 articles.
1. Ozaki K, Ohnishi Y, Iida A et al: Functional SNPs in the lymphotoxin-alpha gene that are associated with susceptibility to myocardial infarction. Nat Genet 2002; 32: 650–654.
2. Messer G, Spengler U, Jung MC et al: Polymorphic structure of the tumor necrosis factor (TNF) locus: an NcoI polymorphism in the first intron of the human TNF-beta gene correlates with a variant amino acid in position 26 and a reduced level of TNF-beta production. J Exp Med 1991; 173: 209–219.
3. Knight JC, Keating BJ, Rockett KA, Kwiatkowski DP : In vivo characterization of regulatory polymorphisms by allele-specific quantification of RNA polymerase loading. Nat Genet 2003; 33: 469–475.
4. Whichelow CE, Hitman GA, Raafat I, Bottazzo GF, Sachs JA : The effect of TNF*B gene polymorphism on TNF-alpha and -beta secretion levels in patients with insulin-dependent diabetes mellitus and healthy controls. Eur J Immunogenet 1996; 23: 425–435.
5. ISFC/WHO: Nomenclature and criteria for diagnosis of ischemic heart disease. Report of the Joint International Society and Federation of Cardiology/World Health Organization task force on standardization of clinical nomenclature. Circulation 1979; 59: 607–609.
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