Intestinal APCs of the endogenous nanomineral pathway fail to express PD-L1 in Crohn’s disease-Reference-Cited by-同舟云学术

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Intestinal APCs of the endogenous nanomineral pathway fail to express PD-L1 in Crohn’s disease

Published:2016-05 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Robertson Jack,Haas Carolin T.,Pele Laetitia C.,Monie Tom P.,Charalambos Charles,Parkes Miles,Hewitt Rachel E.,Powell Jonathan J.

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep26747.pdf

Reference31 articles.

1. Baumgart, D. C. & Sandborn, W. J. Crohn’s disease. Lancet 380, 1590–1605, doi: 10.1016/s0140-6736(12)60026-9 (2012).

2. Hugot, J. P. et al. Association of NOD2 leucine-rich repeat variants with susceptibility to Crohn’s disease. Nature 411, 599–603, doi: 10.1038/35079107 (2001).

3. Hampe, J. et al. A genome-wide association scan of nonsynonymous SNPs identifies a susceptibility variant for Crohn disease in ATG16L1. Nat Genet 39, 207–211, doi: 10.1038/ng1954 (2007).

4. Hedl, M., Li, J., Cho, J. H. & Abraham, C. Chronic stimulation of Nod2 mediates tolerance to bacterial products. Proc Natl Acad Sci USA 104, 19440–19445, doi: 10.1073/pnas.0706097104 (2007).

5. Ramanan, D., Tang, M. S., Bowcutt, R., Loke, P. & Cadwell, K. Bacterial sensor Nod2 prevents inflammation of the small intestine by restricting the expansion of the commensal Bacteroides vulgatus. Immunity 41, 311–324, doi: 10.1016/j.immuni.2014.06.015 (2014).

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