MET is required for the recruitment of anti-tumoural neutrophils-Reference-Cited by-同舟云学术

MET is required for the recruitment of anti-tumoural neutrophils

Published:2015-05-18 Issue:7556 Volume:522 Page:349-353
ISSN:0028-0836
Container-title:Nature
language:en
Short-container-title:Nature

Author:

Finisguerra Veronica,Di Conza Giusy,Di Matteo Mario,Serneels Jens,Costa Sandra,Thompson A. A. Roger,Wauters Els,Walmsley Sarah,Prenen Hans,Granot Zvi,Casazza Andrea,Mazzone Massimiliano

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/nature14407.pdf

Reference35 articles.

1. Gherardi, E., Birchmeier, W., Birchmeier, C. & Vande Woude, G. Targeting MET in cancer: rationale and progress. Nature Rev. Cancer 12, 89–103 (2012)

2. Bertotti, A. et al. Only a subset of Met-activated pathways are required to sustain oncogene addiction. Sci. Signal. 2, ra80 (2009)

3. Lennerz, J. K. et al. MET amplification identifies a small and aggressive subgroup of esophagogastric adenocarcinoma with evidence of responsiveness to crizotinib. J. Clin. Oncol. 29, 4803–4810 (2011)

4. Choueiri, T. K. et al. Phase II and biomarker study of the dual MET/VEGFR2 inhibitor foretinib in patients with papillary renal cell carcinoma. J. Clin. Oncol. 31, 181–186 (2013)

5. Comoglio, P. M., Giordano, S. & Trusolino, L. Drug development of MET inhibitors: targeting oncogene addiction and expedience. Nature Rev. Drug Discov. 7, 504–516 (2008)

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