The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1-Reference-Cited by-同舟云学术

The selective autophagy substrate p62 activates the stress responsive transcription factor Nrf2 through inactivation of Keap1

Published:2010-02-21 Issue:3 Volume:12 Page:213-223
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Komatsu Masaaki,Kurokawa Hirofumi,Waguri Satoshi,Taguchi Keiko,Kobayashi Akira,Ichimura Yoshinobu,Sou Yu-Shin,Ueno Izumi,Sakamoto Ayako,Tong Kit I.,Kim Mihee,Nishito Yasumasa,Iemura Shun-ichiro,Natsume Tohru,Ueno Takashi,Kominami Eiki,Motohashi Hozumi,Tanaka Keiji,Yamamoto Masayuki

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb2021.pdf

Reference54 articles.

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3. Ebato, C. et al. Autophagy is important in islet homeostasis and compensatory increase of beta cell mass in response to high-fat diet. Cell Metab. 8, 325–332 (2008).

4. Jung, H. S. et al. Loss of autophagy diminishes pancreatic beta cell mass and function with resultant hyperglycemia. Cell Metab. 8, 318–324 (2008).

5. Nakai, A. et al. The role of autophagy in cardiomyocytes in the basal state and in response to hemodynamic stress. Nature Med. 13, 619–624 (2007).

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