IL-17 and IL-17-producing cells in protection versus pathology
Author:
Publisher
Springer Science and Business Media LLC
Subject
Energy Engineering and Power Technology,Fuel Technology
Link
https://www.nature.com/articles/s41577-022-00746-9.pdf
Reference245 articles.
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3. Taylor, P. R. et al. Activation of neutrophils by autocrine IL-17A-IL-17RC interactions during fungal infection is regulated by IL-6, IL-23, RORγt and dectin-2. Nat. Immunol. 15, 143–151 (2014).
4. Tamassia, N. et al. A reappraisal on the potential ability of human neutrophils to express and produce IL-17 family members in vitro: failure to reproducibly detect it. Front. Immunol. 9, 795 (2018).
5. Langrish, C. L. et al. IL-23 drives a pathogenic T cell population that induces autoimmune inflammation. J. Exp. Med. 201, 233–240 (2005). This was the first report to describe a population of IL-17-secreting CD4+ T cells (later called TH17 cells) that are distinct from TH1 cells, driven by IL-23, and mediate pathology in an autoimmune disease.
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