Aβ(25–35)-Induced Memory Impairment, Axonal Atrophy, and Synaptic Loss are Ameliorated by M1, A Metabolite of Protopanaxadiol-Type Saponins-Reference-Cited by-同舟云学术

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Aβ(25–35)-Induced Memory Impairment, Axonal Atrophy, and Synaptic Loss are Ameliorated by M1, A Metabolite of Protopanaxadiol-Type Saponins

Published:2003-12-18 Issue:5 Volume:29 Page:860-868
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Tohda Chihiro,Matsumoto Noriaki,Zou Kun,Meselhy Meselhy R,Komatsu Katsuko

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/1300388.pdf

Reference34 articles.

1. Casley CS, Land JM, Sharpe MA, Clark JB, Duchen MR, Canevari L (2002). Amyloid fragment 25–35 causes mitochondrial dysfunction in primary cortical neurons. Neurobiol Dis 10: 258–267.

2. Chan-Palay V (1991). Alterations in the locus coeruleus in dementias of Alzheimer's and Parkinson's disease. Prog Brain Res 88: 625–630.

3. DeKosky ST, Scheff SW (1990). Synapse loss in frontal cortex biopsies in Alzheimer's disease: correlation with cognitive severity. Ann Neurol 27: 457–464.

4. Games D, Adams D, Alessandrini R, Barbour R, Berthelette P, Blackwell C et al (1995). Alzheimer-type neuropathology in transgenic mice overexpressing V717F β-amyloid precursor protein. Nature 373: 523–527.

5. Geddes JW, Anderson KJ, Cotman CW (1986). Senile plaques as aberrant sprout-stimulating structures. Exp Neurol 94: 767–776.

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