Social Interaction Deficits Caused by Chronic Phencyclidine Administration are Reversed by Oxytocin-Reference-Cited by-同舟云学术

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Social Interaction Deficits Caused by Chronic Phencyclidine Administration are Reversed by Oxytocin

Published:2005-03-30 Issue:10 Volume:30 Page:1883-1894
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Lee Paul R,Brady Dana L,Shapiro Robert A,Dorsa Daniel M,Koenig James I

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/1300722.pdf

Reference72 articles.

1. Akbarian S, Sucher NJ, Bradley D, Tafazzoli A, Trinh D, Hetrick WP et al (1996). Selective alterations in gene expression for NMDA receptor subunits in prefrontal cortex of schizophrenics. J Neurosci 16: 19–30.

2. Azorin JM, Spiegel R, Remington G, Vanelle JM, Pere JJ, Giguere M et al (2001). A double-blind comparative study of clozapine and risperidone in the management of severe chronic schizophrenia. Am J Psychiatry 158: 1305–1313.

3. Bale TL, Pedersen CA, Dorsa DM (1995). CNS oxytocin receptor mRNA expression and regulation by gonadal steroids. Adv Exp Med Biol 395: 269–280.

4. Beckmann H, Lang RE, Gattaz WF (1985). Vasopressin–oxytocin in cerebrospinal fluid of schizophrenic patients and normal controls. Psychoneuroendocrinology 10: 187–191.

5. Bernstein HG, Jirikowski GF, Heinemann A, Baumann B, Hornstein C, Danos P et al (2000). Low and infrequent expression of nitric oxide synthase/NADPH-diaphorase in neurons of the human supraoptic nucleus: a histochemical study. J Chem Neuroanatomy 20: 177–183.

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