Glial–Neuronal Interactions are Impaired in the Schizophrenia Model of Repeated MK801 Exposure-Reference-Cited by-同舟云学术

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Glial–Neuronal Interactions are Impaired in the Schizophrenia Model of Repeated MK801 Exposure

Published:2005-12-14 Issue:9 Volume:31 Page:1880-1887
ISSN:0893-133X
Container-title:Neuropsychopharmacology
language:en
Short-container-title:Neuropsychopharmacol

Author:

Kondziella Daniel,Brenner Eiliv,Eyjolfsson Elvar M,Markinhuhta Katarina Ryden,Carlsson Maria L,Sonnewald Ursula

Publisher

Springer Science and Business Media LLC

Subject

Psychiatry and Mental health,Pharmacology

Link

http://www.nature.com/articles/1300993.pdf

Reference47 articles.

1. Baslow MH (2003). N-acetylaspartate in the vertebrate brain: metabolism and function. Neurochem Res 28: 941–953.

2. Brenner E, Kondziella D, Håberg A, Sonnewald U (2005). Impaired glutamine metabolism in NMDA receptor hypofunction induced by MK801. J Neurochem 94: 1594–1603.

3. Brosnan-Watters G, Wozniak DF, Nardi A, Olney JW (1999). Parallel recovery of MK-801-induced spatial learning impairment and neuronal injury in male mice. Pharmacol Biochem Behav 62: 111–122.

4. Burbaeva GS, Boksha IS, Turishcheva MS, Vorobyeva EA, Savushkina OK, Tereshkina EB (2003). Glutamine synthetase and glutamate dehydrogenase in the prefrontal cortex of patients with schizophrenia. Prog Neuro-Psychopharmacol Biol Psychiatry 27: 675–680.

5. Carlsson M (1993). Hypothesis—are the disparate pharmacological profiles of competitive and un-competitive NMDA antagonists due to different baseline activities of distinct glutamatergic pathways? J Neural Transm 94: 1–10.

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