The role of transcriptional factor D-site-binding protein in circadian CCL2 gene expression in anti-Thy1 nephritis-Reference-Cited by-同舟云学术

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The role of transcriptional factor D-site-binding protein in circadian CCL2 gene expression in anti-Thy1 nephritis

Published:2018-03-22 Issue:9 Volume:16 Page:735-745
ISSN:1672-7681
Container-title:Cellular & Molecular Immunology
language:en
Short-container-title:Cell Mol Immunol

Author:

Lu Yang,Mei Yan,Chen Lei,Wu Lingling,Wang Xu,Zhang Yingjie,Fu Bo,Chen Xizhao,Xie Yuansheng,Cai Guangyan,Bai Xueyuan,Li Qinggang,Chen Xiangmei

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Link

http://www.nature.com/articles/s41423-018-0020-4.pdf

Reference33 articles.

1. Liu, X. et al. Change of MAX interactor 1 expression in an anti-Thy1 nephritis model and its effect on mesangial cell proliferation. Cell. Physiol. Biochem. 27, 391–400 (2011).

2. Gao, L. et al. Sublytic complement C5b-9 complexes induce thrombospondin-1 production in rat glomerular mesangial cells via PI3-k/Akt: association with activation of latent transforming growth factor-beta1. Clin. Exp. Immunol. 144, 326–334 (2006).

3. Cantaluppi, V. et al. Endothelial progenitor cell-derived extracellular vesicles protect from complement-mediated mesangial injury in experimental anti-Thy1.1 glomerulonephritis. Nephrol. Dial. Transplant. 30, 410–422 (2015).

4. Aizawa, K. et al. Renoprotective effect of epoetin beta pegol by the prevention of M2 macrophage recruitment in Thy-1 rats. J. Nephrol. 27, 395–401 (2014).

5. Miyasato, K. et al. CD28 superagonist-induced regulatory T cell expansion ameliorates mesangioproliferative glomerulonephritis in rats. Clin. Exp. Nephrol. 15, 50–57 (2011).

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