USP19 suppresses inflammation and promotes M2-like macrophage polarization by manipulating NLRP3 function via autophagy
Author:
Funder
National Natural Science Foundation of China
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Link
https://www.nature.com/articles/s41423-020-00567-7.pdf
Reference52 articles.
1. Saha, S., Shalova, I. N. & Biswas, S. K. Metabolic regulation of macrophage phenotype and function. Immunol. Rev. 280, 102–111 (2017).
2. Murray, P. J. Macrophage polarization. Annu. Rev. Physiol. 79, 541–566 (2017).
3. Kimura, T. et al. Polarization of M2 macrophages requires Lamtor1 that integrates cytokine and amino-acid signals. Nat. Commun. 7, 13130 (2016).
4. Rathinam, V. A. et al. TRIF licenses caspase-11-dependent NLRP3 inflammasome activation by gram-negative bacteria. Cell 150, 606–619 (2012).
5. Hwang, I. et al. Non-transcriptional regulation of NLRP3 inflammasome signaling by IL-4. Immunol. Cell Biol. 93, 591–599 (2015).
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