Point mutations in the gene encoding IP3 receptor subtype 3 cause impairment in T-cell and B-cell immune responses via dysfunctional Ca2+ mobilization
Author:
Publisher
Springer Science and Business Media LLC
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Link
https://www.nature.com/articles/s41423-022-00938-2.pdf
Reference11 articles.
1. Neumann J, Nieuwenhove EV, Terry LE, Staels F, Knebel TR, Welkenhuyzen K, et al. Disrupted Ca2+ homeostasis and immunodeficiency in patients with functional Inositol 1,4,5-trisphosphate receptor subtype 3 defects. Cell Mol Immunol. 2023;20:11–25.
2. Prole DL, Taylor CW. Structure and function of IP3 receptors. Cold Spring Harb Perspect Biol. 2019;11:a035063.
3. Woll KA, Van Petegem F. Calcium-release channels: structure and function of IP3 receptors and ryanodine receptors. Physiol Rev. 2022;102:209–68. https://doi.org/10.1152/physrev.00033.2020
4. Trebak M, Kinet JP. Calcium signalling in T cells. Nat Rev Immunol. 2019;19:154–69.
5. Park YJ, Yoo SA, Kim M, Kim WU. The role of calcium-calcineurin-NFAT signaling pathway in health and autoimmune diseases. Front Immunol. 2020;11:195.
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