Extracellular nucleoprotein exacerbates influenza virus pathogenesis by activating Toll-like receptor 4 and the NLRP3 inflammasome-Reference-Cited by-同舟云学术

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Extracellular nucleoprotein exacerbates influenza virus pathogenesis by activating Toll-like receptor 4 and the NLRP3 inflammasome

Published:2022-04-22 Issue:6 Volume:19 Page:715-725
ISSN:2042-0226
Container-title:Cellular & Molecular Immunology
language:en
Short-container-title:Cell Mol Immunol

Author:

Kim Chang-Ung,Jeong Yu-Jin,Lee Pureum,Lee Moo-Seung,Park Jong-Hwan,Kim Young-Sang,Kim Doo-Jin

Funder

National Research Foundation of Korea

Ministry of Science, ICT and Future Planning

Publisher

Springer Science and Business Media LLC

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Link

https://www.nature.com/articles/s41423-022-00862-5.pdf

Reference50 articles.

1. Couch RB. Seasonal inactivated influenza virus vaccines. Vaccine. 2008;26:D5–9.

2. Takeuchi O, Akira S. Pattern recognition receptors and inflammation. Cell. 2010;140:805–20.

3. Chen X, Liu S, Goraya MU, Maarouf M, Huang S, et al. Host immune response to influenza a virus infection. Front Immunol. 2018;9:320.

4. McAuley JL, Tate MD, MacKenzie-Kludas CJ, Pinar A, Zeng W, et al. Activation of the NLRP3 inflammasome by IAV virulence protein PB1-F2 contributes to severe pathophysiology and disease. PLoS Pathog. 2013;9:e1003392.

5. Ichinohe T, Pang IK, Iwasaki A. Influenza virus activates inflammasomes via its intracellular M2 ion channel. Nat Immunol. 2010;11:404–10.

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