PAR-6–PAR-3 mediates Cdc42-induced Rac activation through the Rac GEFs STEF/Tiam1-Reference-Cited by-同舟云学术

登录注册会员服务联系我们

PAR-6–PAR-3 mediates Cdc42-induced Rac activation through the Rac GEFs STEF/Tiam1

Published:2005-02-20 Issue:3 Volume:7 Page:270-277
ISSN:1465-7392
Container-title:Nature Cell Biology
language:en
Short-container-title:Nat Cell Biol

Author:

Nishimura Takashi,Yamaguchi Tomoya,Kato Katsuhiro,Yoshizawa Masato,Nabeshima Yo-ichi,Ohno Shigeo,Hoshino Mikio,Kaibuchi Kozo

Publisher

Springer Science and Business Media LLC

Subject

Cell Biology

Link

http://www.nature.com/articles/ncb1227.pdf

Reference30 articles.

1. Ohno, S. Intercellular junctions and cellular polarity: the PAR–aPKC complex, a conserved core cassette playing fundamental roles in cell polarity. Curr. Opin. Cell Biol. 13, 641–648 (2001).

2. Macara, I. G. Parsing the polarity code. Nature Rev. Mol. Cell Biol. 5, 220–231 (2004).

3. Shi, S. H., Jan, L. Y. & Jan, Y. N. Hippocampal neuronal polarity specified by spatially localized mPar3/mPar6 and PI 3-kinase activity. Cell 112, 63–75 (2003).

4. Nishimura, T. et al. Role of the PAR-3–KIF3 complex in the establishment of neuronal polarity. Nature Cell Biol. 6, 328–334 (2004).

5. Craig, A. M. & Banker, G. Neuronal polarity. Annu. Rev. Neurosci. 17, 267–310 (1994).

Cited by 329 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Loss of the polarity protein Par3 promotes dendritic spine neoteny and enhances learning and memory;iScience;2024-07

2. Single-cell analysis reveals specific neuronal transition during mouse corticogenesis;Frontiers in Cell and Developmental Biology;2023-11-06

3. Complex relationship among vessel diameter, shear stress and blood pressure controlling vessel pruning during angiogenesis;2023-09-14

4. Loss of the polarity protein Par3 promotes dendritic spine neoteny and enhances learning and memory;2023-09-01

5. Nox1-based NADPH oxidase regulates the Par protein complex activity to control cell polarization;Frontiers in Cell and Developmental Biology;2023-08-11

同舟云学术

1.学者识别学者识别

2.学术分析学术分析

3.人才评估人才评估

"同舟云学术"是以全球学者为主线，采集、加工和组织学术论文而形成的新型学术文献查询和分析系统，可以对全球学者进行文献检索和人才价值评估。用户可以通过关注某些学科领域的顶尖人物而持续追踪该领域的学科进展和研究前沿。经过近期的数据扩容，当前同舟云学术共收录了国内外主流学术期刊6万余种，收集的期刊论文及会议论文总量共计约1.5亿篇，并以每天添加12000余篇中外论文的速度递增。我们也可以为用户提供个性化、定制化的学者数据。欢迎来电咨询！咨询电话：010-8811{复制后删除}0370

www.globalauthorid.com

TOP

Copyright © 2019-2024 北京同舟云网络信息技术有限公司
京公网安备11010802033243号京ICP备18003416号-3