LPS/TLR4 Signaling Enhances TGF-β Response Through Downregulating BAMBI During Prostatic Hyperplasia-Reference-Cited by-同舟云学术

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LPS/TLR4 Signaling Enhances TGF-β Response Through Downregulating BAMBI During Prostatic Hyperplasia

Published:2016-05-31 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

He Yao,Ou Zhenyu,Chen Xiang,Zu Xiongbing,Liu Longfei,Li Yuan,Cao Zhenzhen,Chen Minfeng,Chen Zhi,Chen Hequn,Qi Lin,Wang Long

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep27051.pdf

Reference48 articles.

1. Djavan, B., Margreiter, M. & Dianat, S. S. An algorithm for medical management in male lower urinary tract symptoms. Curr Opin Urol. 21, 5–12 (2011).

2. Shao, R. et al. Epithelial-to-mesenchymal transition and estrogen receptor α mediated epithelial dedifferentiation mark the development of benign prostatic hyperplasia. Prostate. 74, 970–982 (2014).

3. Russo, G. I., Cimino, S. & Morgia, G. Benign prostatic hyperplasia and metabolic syndrome: the expanding evidences of a new disease of aging male. Aging Male. 18, 133–134 (2015).

4. Kramer, G., Mitteregger, D. & Marberger, M. Is benign prostatic hyperplasia (BPH) an immune inflammatory disease? Eur Urol. 51, 1202–1216 (2007).

5. Wang, L., Yang, J. R., Yang, L. Y. & Liu, Z. T. Chronic inflammation in benign prostatic hyperplasia: implications for therapy. Med Hypotheses. 70, 1021–1023 (2008).

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