Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways-Reference-Cited by-同舟云学术

Inversin, the gene product mutated in nephronophthisis type II, functions as a molecular switch between Wnt signaling pathways

Published:2005-04-24 Issue:5 Volume:37 Page:537-543
ISSN:1061-4036
Container-title:Nature Genetics
language:en
Short-container-title:Nat Genet

Author:

Simons Matias,Gloy Joachim,Ganner Athina,Bullerkotte Axel,Bashkurov Mikhail,Krönig Corinna,Schermer Bernhard,Benzing Thomas,Cabello Olga A,Jenny Andreas,Mlodzik Marek,Polok Bozena,Driever Wolfgang,Obara Tomoko,Walz Gerd

Publisher

Springer Science and Business Media LLC

Subject

Genetics

Link

http://www.nature.com/articles/ng1552.pdf

Reference28 articles.

1. Watnick, T. & Germino, G. From cilia to cyst. Nat. Genet. 34, 355–356 (2003).

2. Otto, E.A. et al. Mutations in INVS encoding inversin cause nephronophthisis type 2, linking renal cystic disease to the function of primary cilia and left-right axis determination. Nat. Genet. 34, 413–420 (2003).

3. Mochizuki, T. et al. Cloning of inv, a gene that controls left/right asymmetry and kidney development. Nature 395, 177–181 (1998).

4. Morgan, D. et al. Inversin, a novel gene in the vertebrate left-right axis pathway, is partially deleted in the inv mouse. Nat. Genet. 20, 149–156 (1998).

5. Saadi-Kheddouci, S. et al. Early development of polycystic kidney disease in transgenic mice expressing an activated mutant of the beta-catenin gene. Oncogene 20, 5972–5981 (2001).

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