Dystrophin deficiency promotes leukocyte recruitment in mdx mice-Reference-Cited by-同舟云学术

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Dystrophin deficiency promotes leukocyte recruitment in mdx mice

Published:2019-05-15 Issue:2 Volume:86 Page:188-194
ISSN:0031-3998
Container-title:Pediatric Research
language:en
Short-container-title:Pediatr Res

Author:

Kranig Simon Alexander,Tschada Raphaela,Braun Maylis,Patry Christian,Pöschl Johannes,Frommhold David,Hudalla Hannes

Publisher

Springer Science and Business Media LLC

Subject

Pediatrics, Perinatology and Child Health

Link

http://www.nature.com/articles/s41390-019-0427-3.pdf

Reference40 articles.

1. Knuesel, I. et al. Short communication: altered synaptic clustering of GABAA receptors in mice lacking dystrophin (mdx mice). Eur. J. Neurosci. 11, 4457–4462 (1999).

2. Brunig, I., Suter, A., Knuesel, I., Luscher, B. & Fritschy, J. M. GABAergic terminals are required for postsynaptic clustering of dystrophin but not of GABA(A) receptors and gephyrin. J. Neurosci. 22, 4805–4813 (2002).

3. Constantin, B. Dystrophin complex functions as a scaffold for signalling proteins. Biochim. Biophys. Acta 1838, 635–642 (2014).

4. Evans, N. P., Misyak, S. A., Robertson, J. L., Bassaganya-Riera, J. & Grange, R. W. Immune-mediated mechanisms potentially regulate the disease time-course of duchenne muscular dystrophy and provide targets for therapeutic intervention. PM R. 1, 755–768 (2009).

5. Deconinck, N. & Dan, B. Pathophysiology of duchenne muscular dystrophy: current hypotheses. Pediatr. Neurol. 36, 1–7 (2007).

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