Increased expression of heme oxygenase-1 suppresses airway branching morphogenesis in fetal mouse lungs exposed to inflammation
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology, and Child Health
Link
http://www.nature.com/articles/s41390-019-0588-0.pdf
Reference30 articles.
1. Coalson, J. J. Pathology of new bronchopulmonary dysplasia. Semin. Neonatol. 8, 73–81 (2003).
2. Baraldi, E. & Filippone, M. Chronic lung disease after premature birth. N. Engl. J. Med. 357, 1946–1955 (2007).
3. Choi, C. W. et al. Bronchopulmonary dysplasia in a rat model induced by intra-amniotic inflammation and postnatal hyperoxia: morphometric aspects. Pediatr. Res. 65, 323–327 (2009).
4. Jobe, A. H. et al. Effects of antenatal endotoxin and glucocorticoids on the lungs of preterm lambs. Am. J. Obstet. Gynecol. 182, 401–408 (2000).
5. Jobe, A. H. & Ikegami, M. Antenatal infection/inflammation and postnatal lung maturation and injury. Respir. Res. 2, 27–32 (2001).
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Correction To: Increased expression of heme oxygenase-1 suppresses airway branching morphogenesis in fetal mouse lungs exposed to inflammation;Pediatric Research;2023-06-12
2. Deletion of BACH1 alleviates ferroptosis and protects against LPS-triggered acute lung injury by activating Nrf2/HO-1 signaling pathway;Biochemical and Biophysical Research Communications;2023-02
3. Alteration in branching morphogenesis via YAP/TAZ in fibroblasts of fetal lungs in an LPS-induced inflammation model;Molecular Medicine;2023-01-30
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