Hypothermia increases cold-inducible protein expression and improves cerebellar-dependent learning after hypoxia ischemia in the neonatal rat
Author:
Publisher
Springer Science and Business Media LLC
Subject
Pediatrics, Perinatology and Child Health
Link
https://www.nature.com/articles/s41390-023-02535-z.pdf
Reference88 articles.
1. Fatemi, A., Wilson, M. A. & Johnston, M. V. Hypoxic-ischemic encephalopathy in the term infant. Clin. Perinatol. 36, 835–858 (2009).
2. Graham, E. M., Ruis, K. A., Hartman, A. L., Northington, F. J. & Fox, H. E. A systematic review of the role of intrapartum hypoxia-ischemia in the causation of neonatal encephalopathy. Am. J. Obstet. Gynecol. 199, 587–595 (2008).
3. Askalan, R., Wang, C., Shi, H., Armstrong, E. & Yager, J. Y. The effect of postischemic hypothermia on apoptotic cell death in the neonatal rat brain. Dev. Neurosci. 33, 320–329 (2011).
4. Wassink, G., Gunn, E. R., Drury, P. P., Bennet, L. & Gunn, A. J. The mechanisms and treatment of asphyxial encephalopathy. Front. Neurosci. 8, 40 (2014).
5. Colbourne, F., Grooms, S. Y., Zukin, R. S., Buchan, A. M. & Bennett, M. V. L. Hypothermia rescues hippocampal CA1 neurons and attenuates down-regulation of the AMPA receptor GluR2 subunit after forebrain ischemia. Proc. Natl Acad. Sci. USA. 100, 2906–2910 (2003).
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