PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation
Author:
Publisher
Springer Science and Business Media LLC
Subject
General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry
Link
http://www.nature.com/articles/ncomms13280.pdf
Reference68 articles.
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2. Lamkanfi, M. et al. Inflammasome-dependent release of the alarmin HMGB1 in endotoxemia. J. Immunol. 185, 4385–4392 (2010).
3. Wang, H., Zhu, S., Zhou, R., Li, W. & Sama, A. E. Therapeutic potential of HMGB1-targeting agents in sepsis. Expert Rev. Mol. Med. 10, e32 (2008).
4. Kang, R. et al. HMGB1 in health and disease. Mol. Aspects Med. 40, 1–116 (2014).
5. Moon, J. S. et al. UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis. J. Clin. Invest. 125, 665–680 (2015).
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