PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation-Reference-Cited by-同舟云学术

PKM2-dependent glycolysis promotes NLRP3 and AIM2 inflammasome activation

Published:2016-10-25 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Xie Min,Yu Yan,Kang Rui,Zhu Shan,Yang Liangchun,Zeng Ling,Sun Xiaofang,Yang Minghua,Billiar Timothy R.,Wang Haichao^ORCID,Cao Lizhi,Jiang Jianxin,Tang Daolin

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms13280.pdf

Reference68 articles.

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3. Wang, H., Zhu, S., Zhou, R., Li, W. & Sama, A. E. Therapeutic potential of HMGB1-targeting agents in sepsis. Expert Rev. Mol. Med. 10, e32 (2008).

4. Kang, R. et al. HMGB1 in health and disease. Mol. Aspects Med. 40, 1–116 (2014).

5. Moon, J. S. et al. UCP2-induced fatty acid synthase promotes NLRP3 inflammasome activation during sepsis. J. Clin. Invest. 125, 665–680 (2015).

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