Hyperglycaemia inhibits REG3A expression to exacerbate TLR3-mediated skin inflammation in diabetes-Reference-Cited by-同舟云学术

Hyperglycaemia inhibits REG3A expression to exacerbate TLR3-mediated skin inflammation in diabetes

Published:2016-11-10 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Wu Yelin,Quan Yanchun,Liu Yuanqi,Liu Keiwei,Li Hongquan,Jiang Ziwei,Zhang Tian,Lei Hu,Radek Katherine A.,Li Dongqing,Wang Zhenhua,Lu Jilong,Wang Wang,Ji Shizhao,Xia Zhaofan,Lai Yuping

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms13393.pdf

Reference40 articles.

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2. Lobmann, R. et al. Expression of matrix-metalloproteinases and their inhibitors in the wounds of diabetic and non-diabetic patients. Diabetologia 45, 1011–1016 (2002).

3. Hehenberger, K., Heilborn, J. D., Brismar, K. & Hansson, A. Inhibited proliferation of fibroblasts derived from chronic diabetic wounds and normal dermal fibroblasts treated with high glucose is associated with increased formation of l-lactate. Wound Repair Regen. 6, 135–141 (1998).

4. Wetzler, C., Kampfer, H., Stallmeyer, B., Pfeilschifter, J. & Frank, S. Large and sustained induction of chemokines during impaired wound healing in the genetically diabetic mouse: prolonged persistence of neutrophils and macrophages during the late phase of repair. J. Invest. Dermatol. 115, 245–253 (2000).

5. Rai, N. K., Tripathi, K., Sharma, D. & Shukla, V. K. Apoptosis: a basic physiologic process in wound healing. Int. J. Low. Extrem. Wounds 4, 138–144 (2005).

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