Nlrp12 mutation causes C57BL/6J strain-specific defect in neutrophil recruitment-Reference-Cited by-同舟云学术

Nlrp12 mutation causes C57BL/6J strain-specific defect in neutrophil recruitment

Published:2016-10-25 Issue:1 Volume:7 Page:
ISSN:2041-1723
Container-title:Nature Communications
language:en
Short-container-title:Nat Commun

Author:

Ulland Tyler K.^ORCID,Jain Nidhi,Hornick Emma E.,Elliott Eric I.,Clay Gwendolyn M.,Sadler Jeffrey J.,Mills Kathleen A. M.,Janowski Ann M.,Volk A. Paige Davis,Wang Kai,Legge Kevin L.,Gakhar Lokesh^ORCID,Bourdi Mohammed,Ferguson Polly J.,Wilson Mary E.,Cassel Suzanne L.,Sutterwala Fayyaz S.

Publisher

Springer Science and Business Media LLC

Subject

General Physics and Astronomy,General Biochemistry, Genetics and Molecular Biology,General Chemistry

Link

http://www.nature.com/articles/ncomms13180.pdf

Reference35 articles.

1. Simon, M. M. et al. A comparative phenotypic and genomic analysis of C57BL/6J and C57BL/6N mouse strains. Genome Biol. 14, R82 (2013).

2. Vanden Berghe, T. et al. Passenger mutations confound interpretation of all genetically modified congenic mice. Immunity 43, 200–209 (2015).

3. Mekada, K. et al. Genetic differences among C57BL/6 substrains. Exp. Anim. 58, 141–149 (2009).

4. Schroder, K. & Tschopp, J. The inflammasomes. Cell 140, 821–832 (2010).

5. Wang, L. et al. PYPAF7, a novel PYRIN-containing Apaf1-like protein that regulates activation of NF-kappa B and caspase-1-dependent cytokine processing. J. Biol. Chem. 277, 29874–29880 (2002).

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