Activating somatic mutations outside the SH2-domain of STAT3 in LGL leukemia-Reference-Cited by-同舟云学术

Activating somatic mutations outside the SH2-domain of STAT3 in LGL leukemia

Published:2015-09-30 Issue:5 Volume:30 Page:1204-1208
ISSN:0887-6924
Container-title:Leukemia
language:en
Short-container-title:Leukemia

Author:

Andersson E,Kuusanmäki H,Bortoluzzi S,Lagström S,Parsons A,Rajala H,van Adrichem A,Eldfors S,Olson T,Clemente M J,Laasonen A,Ellonen P,Heckman C,Loughran T P,Maciejewski J P,Mustjoki S

Publisher

Springer Science and Business Media LLC

Subject

Oncology,Cancer Research,Hematology

Link

http://www.nature.com/articles/leu2015263.pdf

Reference15 articles.

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2. Koskela HL, Eldfors S, Ellonen P, van Adrichem AJ, Kuusanmaki H, Andersson EI et al. Somatic STAT3 mutations in large granular lymphocytic leukemia. N Engl J Med 2012; 366: 1905–1913.

3. Fasan A, Kern W, Grossmann V, Haferlach C, Haferlach T, Schnittger S . STAT3 mutations are highly specific for large granular lymphocytic leukemia. Leukemia 2013; 27: 1598–1600.

4. Epling-Burnette PK, Liu JH, Catlett-Falcone R, Turkson J, Oshiro M, Kothapalli R et al. Inhibition of STAT3 signaling leads to apoptosis of leukemic large granular lymphocytes and decreased Mcl-1 expression. J Clin Invest 2001; 107: 351–362.

5. Kucuk C, Jiang B, Hu X, Zhang W, Chan JK, Xiao W et al. Activating mutations of STAT5B and STAT3 in lymphomas derived from gammadelta-T or NK cells. Nat Commun 2015; 6: 6025.

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