The calcium-activated potassium channel KCa3.1 is an important modulator of hepatic injury-Reference-Cited by-同舟云学术

The calcium-activated potassium channel KCa3.1 is an important modulator of hepatic injury

Published:2016-06-29 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Sevelsted Møller Linda,Fialla Annette Dam,Schierwagen Robert,Biagini Matteo,Liedtke Christian,Laleman Wim,Klein Sabine,Reul Winfried,Koch Hansen Lars,Rabjerg Maj,Singh Vikrant,Surra Joaquin,Osada Jesus,Reinehr Roland,de Muckadell Ove B. Schaffalitzky,Köhler Ralf,Trebicka Jonel

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep28770.pdf

Reference53 articles.

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3. Barfod, E. T., Moore, A. L., Roe, M. W. & Lidofsky, S. D. Ca2+-activated IK1 channels associate with lipid rafts upon cell swelling and mediate volume recovery. The Journal of biological chemistry 282, 8984–8993, doi: 10.1074/jbc.M607730200 (2007).

4. Greger, R. et al. The role of K+ channels in colonic Cl- secretion. Comparative biochemistry and physiology. Part A, Physiology 118, 271–275 (1997).

5. Si, H. et al. Impaired endothelium-derived hyperpolarizing factor-mediated dilations and increased blood pressure in mice deficient of the intermediate-conductance Ca2+-activated K+ channel. Circulation research 99, 537–544, doi: 10.1161/01.RES.0000238377.08219.0c (2006).

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