Determinants of interferon β efficacy in patients with multiple sclerosis
Author:
Publisher
Springer Science and Business Media LLC
Subject
Cellular and Molecular Neuroscience,Neurology (clinical)
Link
http://www.nature.com/articles/nrneurol.2011.22.pdf
Reference78 articles.
1. Comi, G. et al. Effect of glatiramer acetate on conversion to clinically definite multiple sclerosis in patients with clinically isolated syndrome (PreCISe study): a randomised, double-blind, placebo-controlled trial. Lancet 374, 1503–1511 (2009).
2. Filippi, M. et al. Interferon-β1a for brain tissue loss in patients at presentation with syndromes suggestive of multiple sclerosis: a randomised, double-blind, placebo-controlled trial. Lancet 364, 1489–1496 (2004).
3. Jacobs, L. D. et al. Intramuscular interferon-β1a therapy initiated during a first demyelinating event in multiple sclerosis. CHAMPS Study Group. N. Engl. J. Med. 343, 898–904 (2000).
4. Kappos, L. et al. Effect of early versus delayed interferon-β1b treatment on disability after a first clinical event suggestive of multiple sclerosis: a 3-year follow-up analysis of the BENEFIT study. Lancet 370, 389–397 (2007).
5. The IFN-β Multiple Sclerosis Study Group and The University of British Columbia MS/MRI Analysis Group. Interferon-β1b in the treatment of multiple sclerosis: final outcome of the randomized controlled trial. Neurology 45, 1277–1285 (1995).
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