An excessive increase in glutamate contributes to glucose-toxicity in β-cells via activation of pancreatic NMDA receptors in rodent diabetes-Reference-Cited by-同舟云学术

An excessive increase in glutamate contributes to glucose-toxicity in β-cells via activation of pancreatic NMDA receptors in rodent diabetes

Published:2017-03-17 Issue:1 Volume:7 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Huang Xiao-Ting,Li Chen,Peng Xiang-Ping,Guo Jia,Yue Shao-Jie,Liu Wei,Zhao Fei-Yan,Han Jian-Zhong,Huang Yan-Hong,Yang-Li ,Cheng Qing-Mei,Zhou Zhi-Guang,Chen Chen,Feng Dan-Dan,Luo Zi-Qiang

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep44120.pdf

Reference44 articles.

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3. Jonas, J. C. et al. Chronic hyperglycemia triggers loss of pancreatic beta cell differentiation in an animal model of diabetes. J Biol Chem 274, 14112–14121 (1999).

4. Bao, Y. et al. Metabonomic variations in the drug-treated type 2 diabetes mellitus patients and healthy volunteers. J Proteome Res 8, 1623–1630, doi: 10.1021/pr800643w (2009).

5. Mihalik, S. J. et al. Metabolomic profiling of fatty acid and amino acid metabolism in youth with obesity and type 2 diabetes: evidence for enhanced mitochondrial oxidation. Diabetes Care 35, 605–611, doi: 10.2337/dc11-1577 (2012).

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