A proteolytic modification of AIM promotes its renal excretion-Reference-Cited by-同舟云学术

A proteolytic modification of AIM promotes its renal excretion

Published:2016-12 Issue:1 Volume:6 Page:
ISSN:2045-2322
Container-title:Scientific Reports
language:en
Short-container-title:Sci Rep

Author:

Yamazaki Tomoko,Sugisawa Ryoichi,Hiramoto Emiri,Takai Ryosuke,Matsumoto Ayaka,Senda Yoshie,Nakashima Katsuhiko,Nelson Peter S.,Lucas Jared M.,Morgan Andrew,Li Zhenghua,Yamamura Ken-ichi,Arai Satoko,Miyazaki Toru

Publisher

Springer Science and Business Media LLC

Subject

Multidisciplinary

Link

http://www.nature.com/articles/srep38762.pdf

Reference33 articles.

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2. Resnick, D., Pearson, A. & Krieger, M. The SRCR superfamily: a family reminiscent of the Ig superfamily. Trends. Biochem. Sci. 19, 5–8 (1994).

3. Sarrias, M. R. et al. The Scavenger Receptor Cysteine-Rich (SRCR) domain: an ancient and highly conserved protein module of the innate immune system. Crit. Rev. Immunol. 24, 1–37 (2004).

4. Yamazaki, T. et al. Circulating AIM as an indicator of liver damage and hepatocellular carcinoma in humans. PLoS One. 9, e109123 (2014).

5. Joseph, S. B. et al. LXR-dependent gene expression is important for macrophage survival and the innate immune response. Cell. 119, 299–309 (2004).

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