Targeting co-stimulatory molecules in autoimmune disease

Author:

Edner Natalie M.ORCID,Carlesso GianlucaORCID,Rush James S.,Walker Lucy S. K.ORCID

Publisher

Springer Science and Business Media LLC

Subject

Drug Discovery,Pharmacology,General Medicine

Reference306 articles.

1. Yu, W. et al. Clonal deletion prunes but does not eliminate self-specific αβ CD8+ T lymphocytes. Immunity 42, 929–941 (2015).

2. Herold, K. C. et al. An anti-CD3 antibody, teplizumab, in relatives at risk for type 1 diabetes. N. Engl. J. Med. 381, 603–613 (2019).

3. Mueller, D. L., Jenkins, M. K. & Schwartz, R. H. An accessory cell-derived costimulatory signal acts independently of protein kinase C activation to allow T cell proliferation and prevent the induction of unresponsiveness. J. Immunol. 142, 2617–2628 (1989).

4. Lesley, R., Kelly, L. M., Xu, Y. & Cyster, J. G. Naive CD4 T cells constitutively express CD40L and augment autoreactive B cell survival. Proc. Natl Acad. Sci. USA 103, 10717–10722 (2006).

5. Nandi, D., Gross, J. A. & Allison, J. P. CD28-mediated costimulation is necessary for optimal proliferation of murine NK cells. J. Immunol. 152, 3361–3369 (1994).

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